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Journal of ENT and Allied Sciences 8(1):p 9-12, January-June 2023. | DOI: NA
Original Article
Hyperpara Thyroidism and its Management
S.P. Subashini, Simrat Kaur, Pooja Jain
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Attribution-Non-commercial 4.0 International (CC BY-NC 4.0)This license enables reusers to distribute, remix, adapt, and build upon the material in any medium or format for noncommercial purposes only, and only so long as attribution is given to the creator.
Journal of ENT and Allied Sciences 8(1):p 9-12, January-June 2023. | DOI: NA
How Cite This Article:
Simrat Kaur, S.P. Subashini, Pooja Jain/Hyperpara Thyroidism and its Management/RFP J ENT Allied Sci 2023;8(1):9–12.Timeline
Received : March 24, 2023
Accepted : May 15, 2023
Published : June 25, 2023
Abstract
Hyperparathyroidism is an increase in parathyroid hormone (PTH) levels in the blood. This occurs from a disorder either within the parathyroid glands (primary hyperparathyroidism) or as response to external stimuli (secondary hyperparathyroidism). Symptoms of hyperparathyroidism are caused by inappropriately normal or elevated blood calcium leaving the bones and flowing into the blood stream in response to increased production of parathyroid hormone. In healthy people, when blood calcium levels are high, parathyroid hormone levels should be low. With long-standing hyperparathyroidism, the most common symptom is kidney stones. Other symptoms may include bone pain, weakness, depression, confusion, and increased urination. Both primary and secondary may result in osteoporosis (weakening of the bones). In 80% of cases, primary hyperparathyroidism is due to a single benign tumor known as a parathyroid adenoma. Most of the remainder are due to several of these adenomas. Very rarely it may be due to parathyroid cancer. Secondary hyperparathyroidism typically occurs due to vitamin D deficiency, chronic kidney disease, or other causes of low blood calcium. The diagnosis of primary hyperparathyroidism is made by finding elevated calcium and PTH in the blood.
References
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- 2. h t t p s : / / w w w . h y p e r p a r a t h y r o i d m d . c o m /hyperparathyroidism/
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Data Sharing Statement
There are no additional data available. All raw data and code are available upon request.
Funding
This research received no funding.
Author Contributions
All authors contributed significantly to the work and approve its publication.
Ethics Declaration
This article does not involve any human or animal subjects, and therefore does not require ethics approval.
Acknowledgements
We would like to express our gratitude to the patients, their families, and all those who have contributed to this study.
Conflicts of Interest
No conflicts of interest in this work.
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Cite this article
Simrat Kaur, S.P. Subashini, Pooja Jain/Hyperpara Thyroidism and its Management/RFP J ENT Allied Sci 2023;8(1):9–12.
Licence:
Attribution-Non-commercial 4.0 International (CC BY-NC 4.0)This license enables reusers to distribute, remix, adapt, and build upon the material in any medium or format for noncommercial purposes only, and only so long as attribution is given to the creator.
| Received | Accepted | Published |
|---|---|---|
| March 24, 2023 | May 15, 2023 | June 25, 2023 |
DOI: NA
Keywords
Vitamin DOsteoporosisParathoromoneRenal RicketsSearch for Similar Articles
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Timeline
| Received | March 24, 2023 |
| Accepted | May 15, 2023 |
| Published | June 25, 2023 |
licence
Attribution-Non-commercial 4.0 International (CC BY-NC 4.0)
This license enables reusers to distribute, remix, adapt, and build upon the material in any medium or format for noncommercial purposes only, and only so long as attribution is given to the creator.
